New Corticosterone CLIA Paper on Genetic KCa3.1 Deficiency in Mice

The calmodulin/calcium-activated K+ channel KCa3.1 is expressed in red and white blood cells, epithelia and endothelia, and possibly central and peripheral neurons. The knowledge about its contribution to neurological functions and behavior is incomplete. The paper examines whether genetic deficiency or pharmacological activation of KCa3.1 change behavior and cerebral monoamine levels in mice.


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